Astrocyte-derived clusterin disrupts glial physiology to obstruct remyelination in mouse models of demyelinating diseases
Abstract Multiple sclerosis (MS) is a debilitating demyelinating disease characterized by remyelination failure attributed to inadequate oligodendrocyte precursor cells (OPCs) differentiation and aberrant astrogliosis.A comprehensive cell atlas reanalysis Vacuum Wine Pumps of clinical specimens brings to light heightened clusterin (CLU) expression